Recent quotes:

Light drinking may be beneficial in type 2 diabetes: Further research needed -- ScienceDaily

The authors found ten relevant RCTs involving 575 participants that were included in this review. Meta-analysis showed that alcohol consumption was associated with reduced triglyceride levels and insulin levels, but had no statistically significant effect on fasting blood glucose levels, glycated haemoglobin (HbA1c, a measure of blood glucose control), or total cholesterol, low density lipoprotein (bad) cholesterol, and high density lipoprotein (good) cholesterol. Subgroup analysis indicated that drinking light to moderate amounts of alcohol decreased the levels of triglycerides (blood fats) and insulin in people with T2DM. Light to moderate drinking was defined by the authors as 20g or less of alcohol per day. This translates to approximately 1.5 cans of beer (330ml, 5% alcohol), a large (200ml) glass of wine (12% alcohol) or a 50ml serving of 40% alcohol spirit (for example vodka/gin).

Ketogenic Diet for Schizophrenia: Clinical Implication

Abnormal glucose and energy metabolism and mitochondrial functioning are emerging as important pathophysiological mechanism in schizophrenia. Therapeutic ketogenic diet shows promise to interfere with these processes resulting in the restoration of normal synaptic communication and alleviation of the devastating psychiatric symptoms. Furthermore, because of the impact of ketogenic diet on systemic metabolism, it is possible that the metabolic features and cardiovascular risk typical of patients with chronic schizophrenia can be addressed by this dietary intervention. However, more research is needed both at preclinical level and in the form of controlled clinical trials before ketogenic diet can take its place in the mainstream of the treatment and management of schizophrenia.

What drives inflammation in type 2 diabetes? Not glucose, says new research - ScienceBlog.com

The team was surprised to find that glycolysis wasn’t driving chronic inflammation. Instead, a combination of defects in mitochondria and elevated fat derivatives were responsible.

It's OK to indulge once in a while, study suggests: The body adapts to occasional short-term overeating: Body focuses on removing glucose to preserve insulin sensitivity in short-term overeating bout -- ScienceDaily

Although the amount of visceral fat that surrounds internal organs increased substantially, short-term overeating did not have a significant effect on the men's weight or fat mass. In addition, fasting levels of blood sugar and C-peptide -- an amino acid the body releases in response to increased production of insulin -- did not change. This finding was surprising because fasting levels of endogenous glucose -- new glucose the body produces in addition to what it has already stored for future use -- increased during the short-term trial. Chronic overeating increased the amount of total body fat and visceral fat as well as post-meal blood sugar and C-peptide levels. However, it did not alter fasting blood sugar levels, endogenous glucose production or the rate of glucose removal from the body (glucose disposal). This may be because the nutrient profile in the long-term trial was consistent with a typical diet and dietary fat percentages did not increase. Long-term overindulgence in fatty foods, instead of more nutritionally balanced foods, may be an important factor that causes rapid changes in blood sugar control.